NIOSH ResourcesĮxposure limits, Respirator Recommendations, First Aid, more… Please contact CDC-INFO (80) if you have any questions regarding carbon monoxide exposure during pregnancy. Hypoxia (severe oxygen deficiency) due to acute carbon monoxide poisoning may result in reversible neurological effects, or it may result in long-term (and possibly delayed) irreversible neurological (brain damage) or cardiological (heart damage) effects.Ĭarbon monoxide exposure can be dangerous during pregnancy for both the mother and the developing fetus. Common symptoms of carbon monoxide exposure are headache, nausea, rapid breathing, weakness, exhaustion, dizziness, and confusion.
As a result, small amounts of carbon monoxide can dramatically reduce hemoglobin’s ability to transport oxygen. Hemoglobin’s binding affinity for carbon monoxide is 300 times greater than its affinity for oxygen. When carbon monoxide is inhaled, it combines with hemoglobin (an iron-protein component of red blood cells), producing carboxyhemoglobin (COHb), which greatly diminishes hemoglobin’s oxygen-carrying capacity.
#Carbon monoxide symptoms in animals portable#
Some examples of this are the following: vehicle exhausts, fuel burning furnaces, coal burning power plants, small gasoline engines, portable gasoline-powered generators, power washers, fire places, charcoal grills, marine engines, forklifts, propane-powered heaters, gas water heaters, and kerosene heaters.Įxposure to carbon monoxide impedes the blood’s ability to carry oxygen to body tissues and vital organs. Consequently, more carbon monoxide, in preference to carbon dioxide, is emitted. Incomplete combustion occurs when insufficient oxygen is used in the fuel (hydrocarbon) burning process. Carbon monoxide (CO) is a colorless, odorless, and toxic gas, which is predominantly produced by incomplete combustion of carbon-containing materials.
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